Year |
Citation |
Score |
2016 |
Zhang X, Ai X, Nakayama H, Chen B, Harris DM, Tang M, Xie Y, Szeto C, Li Y, Li Y, Zhang H, Eckhart AD, Koch WJ, Molkentin JD, Chen X. Persistent increases in Ca(2+) influx through Cav1.2 shortens action potential and causes Ca(2+) overload-induced afterdepolarizations and arrhythmias. Basic Research in Cardiology. 111: 4. PMID 26611208 DOI: 10.1007/S00395-015-0523-4 |
0.302 |
|
2010 |
Brinks H, Boucher M, Gao E, Chuprun JK, Pesant S, Raake PW, Huang ZM, Wang X, Qiu G, Gumpert A, Harris DM, Eckhart AD, Most P, Koch WJ. Level of G protein-coupled receptor kinase-2 determines myocardial ischemia/reperfusion injury via pro- and anti-apoptotic mechanisms. Circulation Research. 107: 1140-9. PMID 20814022 DOI: 10.1161/CIRCRESAHA.110.221010 |
0.311 |
|
2009 |
Chen X, Zhang X, Tang M, Harris D, Li Y, Nakayama H, Zhang H, Berretta R, Eckhart A, Koch W, Molkentin J, Houser S. Persistent Increases In Ca2+ Influx Through Cav1.2 (ICaL)Induce Cardiac Conduction Disturbances And Sarcoplasmic Reticulum Ca2+ Overload To Induce Cardiac Arrhythmia And Sudden Death Biophysical Journal. 96: 221a. DOI: 10.1016/J.Bpj.2008.12.1925 |
0.309 |
|
2008 |
Martini JS, Raake P, Vinge LE, DeGeorge BR, DeGeorge B, Chuprun JK, Harris DM, Gao E, Eckhart AD, Pitcher JA, Koch WJ. Uncovering G protein-coupled receptor kinase-5 as a histone deacetylase kinase in the nucleus of cardiomyocytes. Proceedings of the National Academy of Sciences of the United States of America. 105: 12457-62. PMID 18711143 DOI: 10.1073/pnas.0803153105 |
0.313 |
|
2008 |
Raake PW, Vinge LE, Gao E, Boucher M, Rengo G, Chen X, Degeorge BR, Matkovich S, Houser SR, Most P, Eckhart AD, Dorn GW, Koch WJ. G protein-coupled receptor kinase 2 ablation in cardiac myocytes before or after myocardial infarction prevents heart failure Circulation Research. 103: 413-422. PMID 18635825 DOI: 10.1161/Circresaha.107.168336 |
0.343 |
|
2006 |
Most P, Seifert H, Gao E, Funakoshi H, Völkers M, Heierhorst J, Remppis A, Pleger ST, DeGeorge BR, Eckhart AD, Feldman AM, Koch WJ. Cardiac S100A1 protein levels determine contractile performance and propensity toward heart failure after myocardial infarction. Circulation. 114: 1258-68. PMID 16952982 DOI: 10.1161/CIRCULATIONAHA.106.622415 |
0.307 |
|
2004 |
Most P, Pleger ST, Völkers M, Heidt B, Boerries M, Weichenhan D, Löffler E, Janssen PM, Eckhart AD, Martini J, Williams ML, Katus HA, Remppis A, Koch WJ. Cardiac adenoviral S100A1 gene delivery rescues failing myocardium. The Journal of Clinical Investigation. 114: 1550-63. PMID 15578088 DOI: 10.1172/JCI21454 |
0.313 |
|
2002 |
Keys JR, Greene EA, Koch WJ, Eckhart AD. Gq-coupled receptor agonists mediate cardiac hypertrophy via the vasculature Hypertension. 40: 660-666. PMID 12411459 DOI: 10.1161/01.HYP.0000035397.73223.CE |
0.318 |
|
2002 |
Eckhart AD, Ozaki T, Tevaearai H, Rockman HA, Koch WJ. Vascular-targeted overexpression of G protein-coupled receptor kinase-2 in transgenic mice attenuates β-adrenergic receptor signaling and increases resting blood pressure Molecular Pharmacology. 61: 749-758. PMID 11901213 DOI: 10.1124/Mol.61.4.749 |
0.351 |
|
2001 |
Gaballa MA, Eckhart A, Koch WJ, Goldman S. Vascular β-adrenergic receptor system is dysfunctional after myocardial infarction American Journal of Physiology-Heart and Circulatory Physiology. 280. PMID 11179056 DOI: 10.1152/Ajpheart.2001.280.3.H1129 |
0.338 |
|
Show low-probability matches. |