Henry Efrem Pelish

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2004 Harvard University, Cambridge, MA, United States 
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"Henry Pelish"
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Matthew D. Shair grad student 2004 Harvard
 (The chemistry and biology of secramine.)
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Nitulescu II, Meyer SC, Wen QJ, et al. (2017) Mediator Kinase Phosphorylation of STAT1 S727 Promotes Growth of Neoplasms With JAK-STAT Activation. Ebiomedicine. 26: 112-125
Tahara T, Streit U, Pelish HE, et al. (2017) STAT3 Inhibitory Activity of Structurally Simplified Withaferin A Analogues. Organic Letters
Poss ZC, Ebmeier CC, Odell AT, et al. (2016) Identification of Mediator Kinase Substrates in Human Cells using Cortistatin A and Quantitative Phosphoproteomics. Cell Reports
Pelish HE, Liau BB, Nitulescu II, et al. (2015) Mediator kinase inhibition further activates super-enhancer-associated genes in AML. Nature. 526: 273-6
Marks KM, Park ES, Arefolov A, et al. (2011) The selectivity of austocystin D arises from cell-line-specific drug activation by cytochrome P450 enzymes. Journal of Natural Products. 74: 567-73
Kirchhausen T, Macia E, Pelish HE. (2008) Use of dynasore, the small molecule inhibitor of dynamin, in the regulation of endocytosis. Methods in Enzymology. 438: 77-93
Xu B, Pelish H, Kirchhausen T, et al. (2006) Large scale synthesis of the Cdc42 inhibitor secramine A and its inhibition of cell spreading. Organic & Biomolecular Chemistry. 4: 4149-57
Pelish HE, Ciesla W, Tanaka N, et al. (2006) The Cdc42 inhibitor secramine B prevents cAMP-induced K+ conductance in intestinal epithelial cells. Biochemical Pharmacology. 71: 1720-6
Peterson JR, Lebensohn AM, Pelish HE, et al. (2006) Biochemical suppression of small-molecule inhibitors: a strategy to identify inhibitor targets and signaling pathway components. Chemistry & Biology. 13: 443-52
Pelish HE, Peterson JR, Salvarezza SB, et al. (2006) Secramine inhibits Cdc42-dependent functions in cells and Cdc42 activation in vitro. Nature Chemical Biology. 2: 39-46
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