John D. Crispino
Affiliations: | University of Chicago, Chicago, IL |
Area:
Molecular Biology, BiochemistryGoogle:
"John Crispino"Mean distance: (not calculated yet)
Children
Sign in to add trainee| Rahul S. Bhansali | grad student | (Cell Biology Tree) | |
| Yanfei Xu | grad student | 2005 | Chicago |
| Cindy G. Leung | grad student | 2006 | Chicago |
| Andrew G. Muntean | grad student | 2006 | Chicago |
| Gina T. Kirsammer | grad student | 2007 | Chicago |
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Publications
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| Ayyadevara VSSA, Wertheim G, Gaur S, et al. (2025) DYRK1A inhibition results in MYC and ERK activation rendering KMT2A-R acute lymphoblastic leukemia cells sensitive to BCL2 inhibition. Leukemia |
| Hall T, Mehmood R, Sá da Bandeira D, et al. (2025) Modeling GATA2 deficiency in mice: the R396Q mutation disrupts normal hematopoiesis. Leukemia |
| Jin Q, Harris E, Myers JA, et al. (2024) Disruption of cotranscriptional splicing suggests that RBM39 is a therapeutic target in acute lymphoblastic leukemia. Blood |
| Jin Q, Gutierrez Diaz B, Pieters T, et al. (2022) Oncogenic deubiquitination controls tyrosine kinase signaling and therapy response in acute lymphoblastic leukemia. Science Advances. 8: eabq8437 |
| Zhao X, Bartholdy B, Yamamoto Y, et al. (2022) PU.1-c-Jun interaction is crucial for PU.1 function in myeloid development. Communications Biology. 5: 961 |
| Rammohan M, Harris E, Bhansali RS, et al. (2022) The chromosome 21 kinase DYRK1A: emerging roles in cancer biology and potential as a therapeutic target. Oncogene |
| Bhansali RS, Rammohan M, Lee P, et al. (2021) DYRK1A regulates B cell acute lymphoblastic leukemia through phosphorylation of FOXO1 and STAT3. The Journal of Clinical Investigation. 131 |
| Schieber M, Marinaccio C, Bolanos LC, et al. (2020) FBXO11 is a candidate tumor suppressor in the leukemic transformation of myelodysplastic syndrome. Blood Cancer Journal. 10: 98 |
| Fang C, Rao S, Crispino JD, et al. (2020) Determinants and role of chromatin organization in acute leukemia. Leukemia |
| Laurent AP, Siret A, Ignacimouttou C, et al. (2020) Constitutive activation of RAS/MAPK pathway cooperates with trisomy 21 and is therapeutically exploitable in Down syndrome B-cell leukemia. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research |