Michael Rynkiewicz
Affiliations: | 2015- | Physiology & Biophysics | Boston University, Boston, MA, United States |
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Publications
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Lehman W, Rynkiewicz MJ. (2023) Troponin-I-induced tropomyosin pivoting defines thin-filament function in relaxed and active muscle. The Journal of General Physiology. 155 |
Halder SS, Rynkiewicz MJ, Creso JG, et al. (2023) Mechanisms of pathogenicity in the hypertrophic cardiomyopathy-associated TPM1 variant S215L. Pnas Nexus. 2: pgad011 |
Doran MH, Rynkiewicz MJ, Rassici D, et al. (2023) Conformational changes linked to ADP release from human cardiac myosin bound to actin-tropomyosin. The Journal of General Physiology. 155 |
Doran MH, Rynkiewicz MJ, Pavadai E, et al. (2022) Myosin loop-4 is critical for optimal tropomyosin repositioning on actin during muscle activation and relaxation. The Journal of General Physiology. 155 |
Rynkiewicz MJ, Pavadai E, Lehman W. (2022) Modeling Human Cardiac Thin Filament Structures. Frontiers in Physiology. 13: 932333 |
Pavadai E, Rynkiewicz MJ, Yang Z, et al. (2022) Modulation of cardiac thin filament structure by phosphorylated troponin-I analyzed by protein-protein docking and molecular dynamics simulation. Archives of Biochemistry and Biophysics. 725: 109282 |
Teekakirikul P, Zhu W, Xu X, et al. (2022) Genetic resiliency associated with dominant lethal mutation causing atrial septal defect with high heritability. Cell Reports. Medicine. 3: 100501 |
Sewanan LR, Park J, Rynkiewicz MJ, et al. (2021) Loss of crossbridge inhibition drives pathological cardiac hypertrophy in patients harboring the TPM1 E192K mutation. The Journal of General Physiology. 153 |
Lehman W, Pavadai E, Rynkiewicz MJ. (2021) C-terminal troponin-I residues trap tropomyosin in the muscle thin filament blocked-state. Biochemical and Biophysical Research Communications. 551: 27-32 |
Racca AW, Rynkiewicz MJ, LaFave N, et al. (2020) M8R tropomyosin mutation disrupts actin-binding and filament regulation: the beginning affects the middle and end. The Journal of Biological Chemistry |