Michael Rynkiewicz
Affiliations: | 2015- | Physiology & Biophysics | Boston University, Boston, MA, United States |
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| Creso JG, Gokhan I, Rynkiewicz MJ, et al. (2024) In silico and in vitro models reveal the molecular mechanisms of hypocontractility caused by M8R. Frontiers in Physiology. 15: 1452509 |
| Rynkiewicz MJ, Childers MC, Karpicheva OE, et al. (2024) Myosin's powerstroke transitions define atomic scale movement of cardiac thin filament tropomyosin. The Journal of General Physiology. 156 |
| Barry ME, Rynkiewicz MJ, Pavadai E, et al. (2024) Glutamate 139 of tropomyosin is critical for cardiac thin filament blocked-state stabilization. Journal of Molecular and Cellular Cardiology |
| Lehman W, Rynkiewicz MJ. (2023) Troponin-I-induced tropomyosin pivoting defines thin-filament function in relaxed and active muscle. The Journal of General Physiology. 155 |
| Halder SS, Rynkiewicz MJ, Creso JG, et al. (2023) Mechanisms of pathogenicity in the hypertrophic cardiomyopathy-associated TPM1 variant S215L. Pnas Nexus. 2: pgad011 |
| Doran MH, Rynkiewicz MJ, Rassici D, et al. (2023) Conformational changes linked to ADP release from human cardiac myosin bound to actin-tropomyosin. The Journal of General Physiology. 155 |
| Doran MH, Rynkiewicz MJ, Pavadai E, et al. (2022) Myosin loop-4 is critical for optimal tropomyosin repositioning on actin during muscle activation and relaxation. The Journal of General Physiology. 155 |
| Rynkiewicz MJ, Pavadai E, Lehman W. (2022) Modeling Human Cardiac Thin Filament Structures. Frontiers in Physiology. 13: 932333 |
| Pavadai E, Rynkiewicz MJ, Yang Z, et al. (2022) Modulation of cardiac thin filament structure by phosphorylated troponin-I analyzed by protein-protein docking and molecular dynamics simulation. Archives of Biochemistry and Biophysics. 725: 109282 |
| Teekakirikul P, Zhu W, Xu X, et al. (2022) Genetic resiliency associated with dominant lethal mutation causing atrial septal defect with high heritability. Cell Reports. Medicine. 3: 100501 |