Daryl A. Bosco, Ph.D.
Affiliations: | Neurology | University of Massachusetts Medical School, Worcester, MA, United States |
Area:
Neurodegeneration ALSWebsite:
http://profiles.umassmed.edu/profiles/ProfileDetails.aspx?From=SE&Person=1238Google:
"Daryl Bosco"Bio:
https://profiles.umassmed.edu/display/129995
https://www.boscolab.com/members
http://www.bio.brandeis.edu/prLab/formerMembers.html
Mean distance: 8.73 | S | N | B | C | P |
Parents
Sign in to add mentor| Dorothee Kern | grad student | 2004 | Brandeis | |
| (Investigation of a catalytic role for cyclophilin A in HIV-1 replication.) | ||||
| Jeffery W. Kelly | post-doc | 2003-2005 | Scripps Institute | |
| Gregory A. Petsko | post-doc | 2011 | MGH | |
| Dagmar Ringe | post-doc | 2011 | MGH | |
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Publications
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| Murtas G, Sacchi S, Kumar MS, et al. (2020) Is the primate-specific protein pLG72 affecting SOD1 functionality and superoxide formation? Free Radical Research. 1-25 |
| Bartoletti M, Bosco DA, Da Cruz S, et al. (2019) Phenotypic Suppression of ALS/FTD-Associated Neurodegeneration Highlights Mechanisms of Dysfunction. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 39: 8217-8224 |
| Tischbein M, Baron DM, Lin YC, et al. (2019) The RNA-binding protein FUS/TLS undergoes calcium-mediated nuclear egress during excitotoxic stress and is required for GRIA2 mRNA processing. The Journal of Biological Chemistry |
| Baron DM, Matheny T, Lin YC, et al. (2019) Quantitative proteomics identifies proteins that resist translational repression and become dysregulated in ALS-FUS. Human Molecular Genetics |
| Bosco DA. (2018) Translation dysregulation in neurodegenerative disorders. Proceedings of the National Academy of Sciences of the United States of America |
| Medinas DB, Rozas P, Martínez Traub F, et al. (2018) Endoplasmic reticulum stress leads to accumulation of wild-type SOD1 aggregates associated with sporadic amyotrophic lateral sclerosis. Proceedings of the National Academy of Sciences of the United States of America |
| Sama RR, Fallini C, Gatto R, et al. (2017) ALS-linked FUS exerts a gain of toxic function involving aberrant p38 MAPK activation. Scientific Reports. 7: 115 |
| Peters OM, Cabrera GT, Tran H, et al. (2015) Human C9ORF72 Hexanucleotide Expansion Reproduces RNA Foci and Dipeptide Repeat Proteins but Not Neurodegeneration in BAC Transgenic Mice. Neuron. 88: 902-909 |
| Boopathy S, Silvas TV, Tischbein M, et al. (2015) Structural basis for mutation-induced destabilization of profilin 1 in ALS. Proceedings of the National Academy of Sciences of the United States of America |
| Ward CL, Boggio KJ, Johnson BN, et al. (2014) A loss of FUS/TLS function leads to impaired cellular proliferation. Cell Death & Disease. 5: e1572 |